Nanobacteria/P imPlications
Question:
>Am I the only one who thinks the nanobaclab website seems more like a >hucksterish commercial ad than a scientific presentation?
It’s certainly one of those I’ve-got-a-secret kind of things that one should not trust without the greatest caution, it falls under the "alternative" category at the moment which is bigtime "buyer beware". Absolutely. The thing is, up until the most recent treatments, Amevive and Xanelim, Enbrel and Remicade, none of the "scientific" approaches to psoriasis has been worth much, and some of the "alternative" ones — diet, sunshine, vitamins — have certainly been underemphasised. So, it has paid to keep an open mind. At least the nonobaclab site wasn’t popping up all sorts of disgusting link boxes the whole time, like that chronicfatigue site someone here pointed me at. The Internet: caveat emptor. J.
Response:
>I have just received a call from my GPs office. He is not familiar with this >treatment and would not feel comfortable prescribing it. He would be happy >to refer me to a dermatologist
I suggest you get that referral and discuss it with a dermatologist. The treatment is not generally accepted and I suspect you will not find it effective should you find a doctor willing to try it, but please do your own research on it and discuss it with your physicians. Best regards, Check out our website too–do you love oak furniture? http://www.barnfurniture.com
Response:
>>I have just received a call from my GPs office. He is not familiar with this >treatment and would not feel comfortable prescribing it. He would be happy >to refer me to a dermatologist >I suggest you get that referral and discuss it with a dermatologist. The >treatment is not generally accepted and I suspect you will not find it >effective should you find a doctor willing to try it, but please do your own >research on it and discuss it with your physicians.
I certainly concur on pursuing this only with a dermatologist, not a GP, in the circuit. LadyAndy, do you have any specific info from people who have tried this? Your message sounds rather like you do. J.
Response:
Does this all tie in with the C.A.P.B/antibacterial stuff I mentioned some time ago? Interesting. Bye for now, Peter Finan
Response:
Hi again Cruiser, If you hadn’t said EXLNT. I may have not checked my thoughts on this one. For quite a while i took some anti.s’ for a host of bugs. It was back in 1989. I will go back and look at the protocols and what all i wrote, that i saved in my journal. I have it, still. The journal and a shawdow of the P. At the time i was looking at a fungal infection at the transverse and descending colon. (colon-i-zation.+ fun-gus.. hehe) colon humor… ! My main area for over twenty years. I can name every book, by every kook. And it still bugs me. And i still swear that recolonization of good flora is the best if not the only way to force em out. www.thewholewhey.com (at least read the book obtained from the site… "How to Achieve Maximum Health" by David Webster.) Here is a primer on bugs that ought to bug you. randall… it all comes out in the wash + colons= slightly acidic ph. > How small is this fungus and are there any fungus spores of the size of > viruses?
An emailer sent me this, regards to my threads of searching for a fungus that is the likely cause of prion disease/s. Prion disease, brain echephalopathologies are all about the same in behavior to the animal contracting the disease. Behaviour that probably is meant for the organism to further spread in the future. I am taking the liberty of posting it, for it does answer my question as to size. And it stirs further thoughts. And after reading this emailers message I looked up Mycoplasma organisms for any related to pneumonia since the recent Alzheimer connection with Chlamydia pneumonia. It appears as though Mycoplasma pneumonia exists. Perhaps this organism is the culprit of prion diseases Mycoplasma capricolum (Prokaryota; Bacteria; Tenericutes; Mollicutes; Mycoplasmas; Mycoplasmatales; Mycoplasmataceae) Mycoplasma capricolum Genome Project (Harvard Genome Lab) [S] Mycoplasma capricolum Project : Data Analysis (Harvard Genome Lab) Mycoplasma pneumoniae (Eubacteria; Firmicutes; Low G+C gram-positive bacteria; Mycoplasmas and walled relatives; Mycoplasmatales; Mycoplasmataceae) The Mycoplasma Pneumoniae Genome Project (ZMBH, Heideberg) Mycoplasma pneumoniae (PEDANT, Protein Extraction, Description and ANalysis Tool) (MIPS, Allemagne) [S] KEGG (Kyoto Encyclopedia of Genes and Genomes) : access to genes from their biological functions (Kyoto) : Mycoplasma pneumoniae Genes according to ZMBH. Mycoplasma pneumoniae Genes according to KEGG Search M.pneumoniae database using DBGET Mycobacterium leprae (Eubacteria; Firmicutes; Actinomycetes; Mycobacteria; Mycobacteriaceae; Mycobacterium) Mycobacterium leprae Sequence Data (GTC, Genome Therapeutics Corporation) — quoting an emailer — REITERS SYNDROME/MYCOPLASMS KEY WORDS: Mycoplasm; Chlamydia; Reiters syndrome; Bedsoniae; Mycoplasma; Fiessinger-Leroy-Reitersyndrome; Rickettsia; Ureaplasma; Eaton agent; TWAR; PPLO; OTHER ? THE ORGANISM (Mycoplasmataceae) The story begins with the realization that this "insidious" organism, a Mycoplasm (usually portrayed as Chlamydia) is a melange of microorganisms in nomenclature. It is a viral acting (replicates within living cells) cross between a fungus and a bacterium, very small [(+-)0.3-1.5 micron(um.] with a variably malleable cell wall that probably requires special conditions to do its Invasion and Replication and does not form spores in the usual way, although the two forms ie) 1)"elementary bodies" (infective extracellular state) and 2)"reticulate body"-(intercellular replicating form) may be an early precursor to spore formers. The development cycle takes 24 -48 hr. (from "entry" into the cell to release of "elementary bodies") and may account for the rapid uptake of tetracycline and other antibiotics etc. into it’s cell wall. Outer cell wall resembles that of gram negative bacteria(?). They grow in the cytoplasm of the host cell in a membrane bound vacuole and divide by binary fission. The result of this invasive growth is "The newly formed small particles (elementary bodies) "may" be liberated from the host cell to infect new cells." There appears to be some toxic principle involved. Gram staining is negative or variable. They (?) are rapidly inactivated by heat (60 C. after 10 minutes) and readily maintain their infectivity for years at -50 to -70 C. Freeze drying reduces infectivity although air dried versions remain infective for long periods of time. Lyophilized(?) preparations are also stable for years. They are rapidly inactivated by ether (30 minutes), formalin (0.1% for 24 hrs.), and phenol (0.5% for 24 hrs.). (in vitro or vivo ??) ""The Host- Parasite Relationship- The outstanding biological feature of infection by chlamydiae (type) is the balance that is often reached between host and parasite, resulting in prolonged often lifetime latency. Subclinical infection is the rule-and overt disease the exception in the natural hosts for these agents. Intra species spread, more often leads to disease. Late treatment in clinical cases promotes latency"" { !! } It is very difficult to detect and grow in the laboratory and isolation and differential staining does not provide a conclusive negative test result. There are over 15 serotypes of the Chlamydia organism alone. There are other groups of organisms called (in the early days) mycobacterium and MYCOPLASMAS which are very similar and require better investigative work to be sure of similarities. Organism classification seems to be variable depending on disease symptomology, gram stain typing; morphology, physiology and originating researcher. It appears that serotyping is variable and that these variabilities in antibody titering lead one to believe that there may be a large group of these organisms with many not being detectable with present resources. TRANSMISSION (discussion?) It appears that person to person contact (in it’s various forms) as well as bird to person(Psittacosis) are it’s main routes of transmission (read -contact with an infected (shedding) individual eg) sexual and droplet/dust born inhalation). Other noted transmission is by fingers and flies (Trachoma). "Swimming bath conjunctivitis" has also been implicated via urogenital discharges infecting communal water bodies. There even appears to be familial transmission {Genetic disorders running in families anyone?} One of the principle methods of transmission noted in many books is the birthing process. PROBABLE METHOD OF LONG TERM INFECTIVENESS (subject to discussion) Being a viral acting organism with a potential slime mold exterior, it’s potential for chronic infective states over the long term is well known through the Reiter’s Syndrome disease protocol. The only reliable explanation to this syndrome is that even after treatment (usually with an inappropriate antibiotic and even if a viable one is used for too short a period of time) the mycoplasm is in hiding somewhere, lurking and waiting for "appropriate" conditions to allow for it’s replication/escape and gradual reinfection of other cells. (not too dissimilar to some internet types! (:-) ) Note that for non std transmissions/organisms a protocol similar to Reiter’s would still be valid. {so far} For active clinical diseases, it should be noted that the point of attack is "always" mucosal membranes which sometime presents as inflammation and production of PUSS (that yellow exudate(std)) at the affected site. Other mechanisms probably at work are the slow multiplication inside cells when the body provides weakness; the gradual "colonization" over time where more and more cells are infected thus when the limits of function of a particular system are reached subclinical/clinical "chronic"/acute affects are noticed. Appreciable clinical effects eventually present themselves over time. The type of condition is related to where (which system) the organism has gotten a firm grip. There is also the strong possibility that there are two types of clinical manifestation; {given the organism’s characteristics} 1-Soft Flesh invasion where things go slow…. etc? 2-Circulatory effect…? probably a more reactive process…. THE PSITTACOSIS STORY Prior to about seven years ago this disease was known of; there was a blood test for it; there was also treatment – other antibiotics prior to the development of tetracycline in the early 50’s. Even though the organism was always referred to as a virus {probably the classical definition ala Pasteur – he proved transmission (the existence of several organisms) but could not see, isolate nor identify them} the source(s) (birds) and the mode of transmission (inhalation of droppings {?} were known. IMMUNE REACTIONS There is evidence that cell mediated immune reaction is the body’s defense mechanism against this INSIDIOUS attack as well as the obvious macrophage reaction at massive attack on initial attempts at penetration at the mucosa. {There may be variation depending on which form incites which immune reaction} -Shows Compliment fixing reactivity (psittacosis) -surrounding with calcium complex (clogged arterials) -macrophage sensitivity (puss formation) -OTHER {????} KNOWN CLINICAL MANIFESTATIONS 1 - NSU-NGU(non specific/ghonorea urithritis) (C. trachomatis) 2 - Psittacosis (C. psittaci) – secondary complications include hepatitis, myocarditis, endocarditus, meningitis, mild flu like disease, asymptomatic infection(?) 3 - PNEUMONIA (C. pneumoniae)(isolated 1965)- similar to Mycoplasma pneumoniae – two types of antibody tests 1) Primary infection 2) reinfection/recurrence(6-10% of pneumonias - leads to Bronchitis/Pharyngitis; at age 20 1/2 of people tested have antibodies to the organism 4 … read more »
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>Naw, sounds like something a german scientist came up with >as to armoring and psyches. What was his name? It was the orgonne >theory something? Oh well. >Maybe now that he is dead and gone, we will give him some small >bit of glory. Naw. Screw his stupid horny weird ness. ( It was Reich?)
Yep. Oy. <g> J.
Response:
Hi Crz’er, Yep, and the bugs are real smart. They will form a protective barrier around the inner bugs so as to protect the continuation of the species. Gosh, bugs are altruistic. And most people would have thought it was every bug for his or her self. I wonder how many genes we share with *them*. Maybe i can go over to the lifescore people and see if we can have some of us, go to a nurse and draw some blood to see if that 11 of 13 isn’t more like 90 something outa 100. randall… nano nano… yipes babytalk.. – Hide quoted text — Show quoted text – > This is just the sort of information I have been looking for. Excellent. > When I had a parasitic infection many years ago, I was prescribed > Doxycycline. My nails began to clear up! When I explained this side effect > to my doctor, he told me he was not about to prescribe antibiotics to treat > psoriasis. > Well, now I learn that when tested 11 of 13 psoriatics had nanobacteria. Now > here is the kicker. Tetracycline kills active nanobacteria which have not > gone dormant in a calcium shell. Doxycycline is a tetracycline antibiotic. > However, once you stop treatment , the protected nanobacteria must be able > to come out of their shells and re-infect. > I have already faxed my doctor some info from the WEB page and told him I > want to be tested for nanobacteria and if positive, try the treatment. The > treatment is supposed to strip the calcium protection and kill all the > nanobacteria, even the protected ones. The thing is that this treatment must > use tetracycline in combination with some other agent, both of which are > already FDA approved. > Cruiser > Hi, > Most of this site is into the calcification of > arteries as its more target rich then P research. > Yet they found 11 of 13 P’s with nanobacteria. > A local company is doing a study at www.lifescore.com > with clearing plaque in the arteries. I think they > are using tetracycline as the antibiotic to clear the > nanobacteria.. > Here is another area to poke around in.. > www.nanobaclabs.com > and a blurb from their url… > Introduction: Nanobacteria are newly-found autonomously replicating > particles that have been detected in mineral stones like in kidney > stones (1). The nature of nanonacteria has been the focus of intensive > research for a few years, and also scientific debate has been carried > out, especially, whether to name the particles as nanobacteria or > nanoparticles since their properties are clearly distinct from those > of the bacteria in general. Nanoparticles have been detected also in > fetal bovine serum as well as in human serum. In dermatology, there > are numerous skin diseases with unknown etiology, and infective origin > has been suggested. Thus, nanoparticles were studied in various > dermatological diseases. > Methods: Blood samples and skin biobsy specimens from various skin > disorders (65 patients total) were collected, and the samples were > analyzed in the Department of Biochemistry at the University of > Kuopio. The methodology used is described in detail in the > presentation of Dr. Kuronen in this Symposium. > Results: 12% (3 of 26 healthy controls; 8 male, 18 female, age 22-48 > yrs) showed nano-positivity on serum culture. Similar percentage (14%, > i.e. 1/7 controls) was observed when analyzed for nano-specific IgG, > IgM, and culture IF-study. From patients, also skin biopsies were > obtained from healthy and diseased skin. When combining all the > different analysis results, psoriatics showed positivity in 11 of 13 > patients (85%), patients with lichen ruber planus in 6 of 6 (100%), > patients with nummular or other eczema in 6 of 6 patients (100%), and > patients with urticaria and nonspecific pruritus in 8 of 9 patients > (89%). The corres-ponding serum culture IF-positivities were 53%, 67%, > 60% and 56%, respectively. Three of four patients with prurigo > nodularis were culture-IF-positive. > Discussion: Nanoparticles are clearly associated in various > inflammatory skin diseases, such as psoriasis, lichen ruber planus, > nummular eczema and other nonspecific dermatitis, and also urticaria > and nonspecific pruritus. From these results, it is not known whether > nanoparticles have a role as primary or secondary factor in these > diseases. A few patients studied that nanoparticles may have a role in > various skin diseases, such as amyloidosis and prurigo nodularis (that > is a severe inflammatory nodular itchy dermatosis). The presence of > nanoparticles were observed in a few patients with skin diseases such > as scleroderma and lichen sclerosus, pyoderma gangrenosum and Sezary > syndrome (a rare malignant T cell lymphoma). These patients were too > few to draw a definite conclusion. More studies are needed to > elucidate the role on nanoparticles in these diseases. > So? Or are the above wiggle words "clearly associated" to weak for us? > Who has or had chymidia and or kidney stones prior to P, as a > initiating event? > randall… keep your powder dry. Revenge of the nano bacs.
Response:
>> www.nanobaclabs.com … >I have already faxed my doctor some info from the WEB page and told him I >want to be tested for nanobacteria and if positive, try the treatment. The >treatment is supposed to strip the calcium protection and kill all the >nanobacteria, even the protected ones. The thing is that this treatment must >use tetracycline in combination with some other agent, both of which are >already FDA approved.
Well, I’d sure like to hear how it works for some brave volunteers. An undisclosed mix of prescription drugs? Um, … I’d skipped over the web site in reading Randall’s earlier post. It is interesting. J.
Response:
I have just received a call from my GPs office. He is not familiar with this treatment and would not feel comfortable prescribing it. He would be happy to refer me to a dermatologist however. I told him that I would wait on the results of the nail fungus test he sent out recently. If the results are negative, I will follow-up on this test and subsequent treatment. Ron
– Hide quoted text — Show quoted text ->> www.nanobaclabs.com > … >I have already faxed my doctor some info from the WEB page and told him I >want to be tested for nanobacteria and if positive, try the treatment. The >treatment is supposed to strip the calcium protection and kill all the >nanobacteria, even the protected ones. The thing is that this treatment must >use tetracycline in combination with some other agent, both of which are >already FDA approved. > Well, I’d sure like to hear how it works for some brave volunteers. > An undisclosed mix of prescription drugs? Um, … > I’d skipped over the web site in reading Randall’s earlier post. > It is interesting. > J.
Response:
Hi Maddie, Thanks, this helps. I have been looking at pneumococcal and Chlamydia bacteria for awhile now. I just wonder if its not a endotoxin or nanobacteria from them that causes the p ruckus. A major trigger, that i’ve overlooked. That sits dormant like shingles (herpes) or some other stressor. Good work. Maybe its even more insidious. waiting for some protein to take a longer then normal transit and it makes its living during the final hours of colon transit. Naw, sounds like something a german scientist came up with as to armoring and psyches. What was his name? It was the orgonne theory something? Oh well. Maybe now that he is dead and gone, we will give him some small bit of glory. Naw. Screw his stupid horny weird ness. ( It was Reich?) God i’m good. If i don’t think of it right away, then give my long term brain cells a crack at it and bingo. I’m so smart that i’ll go google and check it. What a idiot. I should go back and look at bacteria. Oh Poop. P is the most anal disease. I do de clare. thanks any way margret. I feel this is a good venue for research. randall… on the sly.. for bacteria that defecate. – Hide quoted text — Show quoted text – > I saw this on the BBC Health newsite about an enzyme being used to kill > bacteria. > "Scientists have discovered an enzyme which could be a "natural born killer" > of infections – and it could be delivered through a nasal spray. > It could be the secret weapon scientists have been seeking for decades > because of the growth of antibiotic-resistant infections" > more on page below > http://news.bbc.co.uk/hi/english/health/newsid_1696000/1696060.stm > >So? Or are the above wiggle words "clearly associated" too weak for us? > Yup. > "More study needed". > J. > — > Outgoing mail is certified Virus Free. > Checked by AVG anti-virus system (http://www.grisoft.com).
Response:
This is just the sort of information I have been looking for. Excellent. When I had a parasitic infection many years ago, I was prescribed Doxycycline. My nails began to clear up! When I explained this side effect to my doctor, he told me he was not about to prescribe antibiotics to treat psoriasis. Well, now I learn that when tested 11 of 13 psoriatics had nanobacteria. Now here is the kicker. Tetracycline kills active nanobacteria which have not gone dormant in a calcium shell. Doxycycline is a tetracycline antibiotic. However, once you stop treatment , the protected nanobacteria must be able to come out of their shells and re-infect. I have already faxed my doctor some info from the WEB page and told him I want to be tested for nanobacteria and if positive, try the treatment. The treatment is supposed to strip the calcium protection and kill all the nanobacteria, even the protected ones. The thing is that this treatment must use tetracycline in combination with some other agent, both of which are already FDA approved. Cruiser
– Hide quoted text — Show quoted text -> Hi, > Most of this site is into the calcification of > arteries as its more target rich then P research. > Yet they found 11 of 13 P’s with nanobacteria. > A local company is doing a study at www.lifescore.com > with clearing plaque in the arteries. I think they > are using tetracycline as the antibiotic to clear the > nanobacteria.. > Here is another area to poke around in.. > www.nanobaclabs.com > and a blurb from their url… > Introduction: Nanobacteria are newly-found autonomously replicating > particles that have been detected in mineral stones like in kidney > stones (1). The nature of nanonacteria has been the focus of intensive > research for a few years, and also scientific debate has been carried > out, especially, whether to name the particles as nanobacteria or > nanoparticles since their properties are clearly distinct from those > of the bacteria in general. Nanoparticles have been detected also in > fetal bovine serum as well as in human serum. In dermatology, there > are numerous skin diseases with unknown etiology, and infective origin > has been suggested. Thus, nanoparticles were studied in various > dermatological diseases. > Methods: Blood samples and skin biobsy specimens from various skin > disorders (65 patients total) were collected, and the samples were > analyzed in the Department of Biochemistry at the University of > Kuopio. The methodology used is described in detail in the > presentation of Dr. Kuronen in this Symposium. > Results: 12% (3 of 26 healthy controls; 8 male, 18 female, age 22-48 > yrs) showed nano-positivity on serum culture. Similar percentage (14%, > i.e. 1/7 controls) was observed when analyzed for nano-specific IgG, > IgM, and culture IF-study. From patients, also skin biopsies were > obtained from healthy and diseased skin. When combining all the > different analysis results, psoriatics showed positivity in 11 of 13 > patients (85%), patients with lichen ruber planus in 6 of 6 (100%), > patients with nummular or other eczema in 6 of 6 patients (100%), and > patients with urticaria and nonspecific pruritus in 8 of 9 patients > (89%). The corres-ponding serum culture IF-positivities were 53%, 67%, > 60% and 56%, respectively. Three of four patients with prurigo > nodularis were culture-IF-positive. > Discussion: Nanoparticles are clearly associated in various > inflammatory skin diseases, such as psoriasis, lichen ruber planus, > nummular eczema and other nonspecific dermatitis, and also urticaria > and nonspecific pruritus. From these results, it is not known whether > nanoparticles have a role as primary or secondary factor in these > diseases. A few patients studied that nanoparticles may have a role in > various skin diseases, such as amyloidosis and prurigo nodularis (that > is a severe inflammatory nodular itchy dermatosis). The presence of > nanoparticles were observed in a few patients with skin diseases such > as scleroderma and lichen sclerosus, pyoderma gangrenosum and Sezary > syndrome (a rare malignant T cell lymphoma). These patients were too > few to draw a definite conclusion. More studies are needed to > elucidate the role on nanoparticles in these diseases. > So? Or are the above wiggle words "clearly associated" to weak for us? > Who has or had chymidia and or kidney stones prior to P, as a > initiating event? > randall… keep your powder dry. Revenge of the nano bacs.
Response:
Hi, Most of this site is into the calcification of arteries as its more target rich then P research. Yet they found 11 of 13 P’s with nanobacteria. A local company is doing a study at www.lifescore.com with clearing plaque in the arteries. I think they are using tetracycline as the antibiotic to clear the nanobacteria.. Here is another area to poke around in.. www.nanobaclabs.com and a blurb from their url… Introduction: Nanobacteria are newly-found autonomously replicating particles that have been detected in mineral stones like in kidney stones (1). The nature of nanonacteria has been the focus of intensive research for a few years, and also scientific debate has been carried out, especially, whether to name the particles as nanobacteria or nanoparticles since their properties are clearly distinct from those of the bacteria in general. Nanoparticles have been detected also in fetal bovine serum as well as in human serum. In dermatology, there are numerous skin diseases with unknown etiology, and infective origin has been suggested. Thus, nanoparticles were studied in various dermatological diseases. Methods: Blood samples and skin biobsy specimens from various skin disorders (65 patients total) were collected, and the samples were analyzed in the Department of Biochemistry at the University of Kuopio. The methodology used is described in detail in the presentation of Dr. Kuronen in this Symposium. Results: 12% (3 of 26 healthy controls; 8 male, 18 female, age 22-48 yrs) showed nano-positivity on serum culture. Similar percentage (14%, i.e. 1/7 controls) was observed when analyzed for nano-specific IgG, IgM, and culture IF-study. From patients, also skin biopsies were obtained from healthy and diseased skin. When combining all the different analysis results, psoriatics showed positivity in 11 of 13 patients (85%), patients with lichen ruber planus in 6 of 6 (100%), patients with nummular or other eczema in 6 of 6 patients (100%), and patients with urticaria and nonspecific pruritus in 8 of 9 patients (89%). The corres-ponding serum culture IF-positivities were 53%, 67%, 60% and 56%, respectively. Three of four patients with prurigo nodularis were culture-IF-positive. Discussion: Nanoparticles are clearly associated in various inflammatory skin diseases, such as psoriasis, lichen ruber planus, nummular eczema and other nonspecific dermatitis, and also urticaria and nonspecific pruritus. From these results, it is not known whether nanoparticles have a role as primary or secondary factor in these diseases. A few patients studied that nanoparticles may have a role in various skin diseases, such as amyloidosis and prurigo nodularis (that is a severe inflammatory nodular itchy dermatosis). The presence of nanoparticles were observed in a few patients with skin diseases such as scleroderma and lichen sclerosus, pyoderma gangrenosum and Sezary syndrome (a rare malignant T cell lymphoma). These patients were too few to draw a definite conclusion. More studies are needed to elucidate the role on nanoparticles in these diseases. So? Or are the above wiggle words "clearly associated" to weak for us? Who has or had chymidia and or kidney stones prior to P, as a initiating event? randall… keep your powder dry. Revenge of the nano bacs.
Response:
>I saw this on the BBC Health newsite about an enzyme being used to kill >bacteria. >"Scientists have discovered an enzyme which could be a "natural born killer" >of infections – and it could be delivered through a nasal spray. >It could be the secret weapon scientists have been seeking for decades >because of the growth of antibiotic-resistant infections" >http://news.bbc.co.uk/hi/english/health/newsid_1696000/1696060.stm
Very interesting. Might be a good inhaler/spray if you get a sore throat, or a good topical if you get so much as an infected pimple. OTOH, if it turns out to burst healthy human cells as well, it will require some serious work to turn into a useful drug. Thanks. J.
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>So? Or are the above wiggle words "clearly associated" too weak for us?
Yup. "More study needed". J.
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I saw this on the BBC Health newsite about an enzyme being used to kill bacteria. "Scientists have discovered an enzyme which could be a "natural born killer" of infections – and it could be delivered through a nasal spray. It could be the secret weapon scientists have been seeking for decades because of the growth of antibiotic-resistant infections" more on page below http://news.bbc.co.uk/hi/english/health/newsid_1696000/1696060.stm
>So? Or are the above wiggle words "clearly associated" too weak for us? > Yup. > "More study needed". > J.
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